Search
Showing results for "early lung health"
This study aimed to evaluate and compare the effectiveness of two brief written anti-stigma resources.
To investigate specific child and parental factors associated with increased vulnerability to substantiated child maltreatment.
To investigate the prevalence, trends, and characteristics of maltreatment and assault related hospital admissions and deaths among children;
The first large scale scientific evaluation of group-based positive parenting programs has found that the intervention reduces behavioural problems
Associate Professor Pradeep Rao called for more hope among clinicians and young people alike when accessing services.
This study examines the association between PAE in the general antenatal population and child neurodevelopment at 2 years, accounting for relevant factors.
Tuberculosis (TB) is the leading infectious cause of death globally, with approximately three million cases remaining undetected, thereby contributing to community transmission. Understanding the spatial distribution of undetected TB in high-burden settings is critical for designing and implementing geographically targeted interventions for early detection and control.
Dynamic cellular and molecular adaptations in early life significantly impact health and disease. Upon birth, newborns are immediately challenged by their environment, placing urgent demands on the infant immune system.
Reducing the incidence and prevalence of standard modifiable cardiovascular risk factors (SMuRFs) is critical to tackling the global burden of coronary artery disease (CAD). However, a substantial number of individuals develop coronary atherosclerosis despite no SMuRFs. SMuRFless patients presenting with myocardial infarction have been observed to have an unexpected higher early mortality compared to their counterparts with at least 1 SMuRF.
Cells undergo a major epigenome reconfiguration when reprogrammed to human induced pluripotent stem cells (hiPS cells). However, the epigenomes of hiPS cells and human embryonic stem (hES) cells differ significantly, which affects hiPS cell function. These differences include epigenetic memory and aberrations that emerge during reprogramming, for which the mechanisms remain unknown.